EFFECTS OF CARBON MONXIDE EXPOSURE ON HAEMATOLOGICAL PARAMETERS

Erythrocytes, Haemoglobin and packed cell volume:
Reports have it that carbon monoxide displaces oxygen (O2) in the bloodstream thus depriving the heart, brain and other vital organs of appropriate blood supply. Studies has also recorded that carbon monoxide binds to red blood cells 200-240 times more than the oxygen. The United State Department of Labour’s Occupational Safety and Health Association (OSHA; 2009)
classifies CO as an asphyxiant (which CO means it displaces O2) and causes symptoms which invariably leads to death. when CO gas is inhaled it prevents human blood from carrying enough life-maintaining O2 to the body and brain i.e. why it is lethal at high level. Even at low levels, CO is dangerous especially for people with pre-existing medical conditions such as heart disease, asthma and bronchitis among others. If CO is present in the lung, fthe normal sequence is altered by binding with iron in haemoglobnin to form carboxyhaemoglin, a complex similar to oxyhaemoglobin which is a more stable compound.

When it reaches the cells, it has little tendency to break apart but continues to circulate in the bloodstream in its bound form. As a result of this, the cells are unable to obtain the O2 they need for energy production and symptoms of CO poisoning begins to appear. This produces a wide range of effects on health; one of the major effects is that the high concetntration of the Co reduces the level of oxygen that is carried to the brain. A study carried out on the relationship among cigarette smoking, haemoglin (Hb) concentration and carboxyhaemoglobin concentration showed a positive correction between cigarette consumption and packed cell volume. The values of PCV and other subnormal parameters such as MCHC were stastically higher than in males who do not smoke (Henrik and Leif; 2009).

However, there were significant (p<0.05) means legless and carboxyhaemoglobin levels increased progressively with the number of cigarette consumed per day and it was alos suggest that cigarette smoking seems to have a generalized upward shift of the haemoglobin distribution curve (Nordenberg et al; 1990). Oxygen majority binds to Hb leading to formation of oxyhaemoglobin and thus the oxygenated blood passes from the lungs through the bloodstream to all the tissues of the body. Carboxyhaemogblin compound formed also has direct effect on the blood vessels of the body-thus causing them to be leaky, this is majority seen in the brain (Henry; 1999). 

The result of a certain study carried out showed and suggested that those smokers in the courses of smoking inhale CO and this typically was significant with low-grade CO and other mild respiratory. A raised haematocrit reflects haemo-concentration and this may relative due to reduced plasma voume or absolute (increased red blood cell mass). A study has it that reduced plasma volunme can occur due to dehydration and also acute pancreatitis, loss water, increased red cell mass, smoking, altitude, tumor (hematoma) are some of the factors that can affect PCV. Individuals with an increased PCV may be associated with an increase in thrombotic events and cardiovascular mortality (Lowe; 1999).
Share on Google Plus

Declaimer - Unknown

The publications and/or documents on this website are provided for general information purposes only. Your use of any of these sample documents is subjected to your own decision NB: Join our Social Media Network on Google Plus | Facebook | Twitter | Linkedin

READ RECENT UPDATES HERE