ENVIRONMENTAL TOXICOLOGY


DEFINITION
* Toxicology is the study of the harmful effects of chemicals on biologic systems.
An interdisciplinary science with input from biochemistry, physiology, pathology, pharmacology, chemistry and public health.
*  Environmental toxicology focuses on exposure to toxic substances in the general environment- air, food and water and also the occupational environment.
CLASSIFICATION
*  Physical state
*  Airborne toxics – dusts, fumes, mists, vapour
*  Liquids

*  solid
*  Chemical groups
*  Metals
*  Solvents eg toluene, xylene, benzene etc
*  Acids
*  Alkalis
>  Organophosphorous compounds
*  According to target organs
*  Liver                    Lungs
*  Kidney                 Skin
*  Blood                   Nervous system
ABSORBTION
*  Respiratory – dusts, fumes, vapours etc
   water soluble toxins exert immediate effects in the respiratory system.
Less soluble substances dissolve more slowly producing acute pneumonitis and pulmonary oedema.
*  Asphyxiants – interrupt the supply of oxygen. Simple asphyxiants eg N2 displaces O2 in the air. Chemical asphyxiants – CO combines with Hb to form carboxyhaemoglobin.
      DISTRIBUTION
  •   Destination of toxins determined by
n  Ability to cross membrane barrier
n  Concentration in the blood
n  Affinity for particular body compartments
  •   Some substances are transported by binding.
*  Haem – CO
*  RBC – Pb
*  Albumin – Hg
*  Globulins – Fe, Zn
STORAGE
*  At site of action eg CO
*  Affinity eg where naturally occurring element with similar properties exist _ Pb and Ca.
*  Fat soluble substance may be stored in adipose tissue eg DDT, dieldrin.
Toxin can be mobilized from the stores.
BIOLOGICAL EFFECTS
* TYPES OF EFFECTS
*  Acute/ chronic toxicity
*  Allergy
*  Carcinogenicity
*  Reproductive and developmental effects
Effects are usually dose respondent
                                Dose response curve
                                LD50 – a measure of potency
METABOLISM
n  Breakdown of toxins to simpler molecules
n  Oxidation
n  Reduction
n  Conjugation
n  Hydrolysis
EXCRETION
*  Routes
*  Kidney
*  Biliary tract
*  Lungs
*  Breast milk
*  Sweat
*  Hair
*  Saliva
EXAMPLES OF TARGET ORGANIC RESPONSES
*  Liver
*  Hepatocellular damage eg ethyl alcohol, cyclophosphamide
*  Carcinogenic effects
- Aflatoxin
- Vinyl chloride monomers
*  Kidney
-  Tubular damage – heavy metals Pb, Cd and Hg. paraquat
*  Blood
-  Pb inhibits production of haemoglobin
       1. inhibits the formation of globin moiety
    2.  red cell membrane fragility
-  Benzene and radiation  a- plastic anaemia
                                    1.      leukaemia
*  Lungs
*  Acute responses. Irritants, allergies
*  Chronic responses. Fibrogenic dusts
*  Carcinogenic – asbestos, nickel
*  CNS
*  Acute response – solvents eg acetone, toluene,    headache, dizziness, confusion, narcosis
*  Chronic
*  Pb – foot or wrist develop- motor neuron damage
*  Mn – damage to cerebellum- parkinsonism
*  Organophosphates – inhibits acetyl cholinesterase    and carbonates .
    ACH accumulates at parasympathetic nerve ending.
    increased red cell cholinesterase.
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