PATHOGENESIS OF TUBERCULOSIS

Transmission is by air droplet. Sneezing, coughing or talking releases about 40,000 droplet (0.5-5 micrometer in diameter), each capable of infection. When inhaled by unsuspecting person, process of infection starts. The bacillus enters the lung to cause a non-specific inflammatory response resulting  in Ghon’s focus and regional node inflammation (primary complex). Primary complex heals spontaneously with fibrosis and calcification forming a granuloma where the bacillus persists in a dormant form that can be reactivated any time.

          Individual immunity is important in the response to infection. About 90% of those infected have asymptomatic latent infection while 10% may progress from latent infection to the disease. Adult pulmonary tuberculosis may therefore occur by reactivation of latent bacilli or exogenous re-infection.
          Body develops certain level of cell mediated immunity that protects the individual. This immunity is destroyed by HIV/AIDS leading to reactivation, hence increase of tuberclosis cases. Destruction of lung paranchyma and cavitation (Tubercle) is important feature of the adult tuberculosis. This is also called cell death (Necrosis) (K. Parks 2009).
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