MORPHOLOGY AND IDENTIFICATION OF TUBERCULOSIS


A.  Typical organism: In tissue, tubercle bacilli are thin straight rods measuring about 0.4 x 3 um. On artificial media,  coccoid and filamentious forms are seen with variable  morphology from one specie to another. Mycobacterium can be classified as either gram- positive or gram- negative. Once stained by basic dyes they cannot be decolorized by alcohol regardless of treatment with iodine. True tubercle bacilli are characterized by “acid- fastness” which means 95% ethyl alcohol containing 3% hydrochloric acid can quickly decolorize all bacteria except the mycobacterium. Acid fastness depends on the integrity of the waxy envelope.
The ziehl-Neelsen technique of staining is employed for identification of acid fast – bacteria (Jawetz et al, 1998).

B Culture: The media for primary culture of mycobacterium should include a non selective medium and a selective medium. Selective media contain antibiotics to prevent the overgrowth of contaminating bacteria and fungi (Burrel, 2005). 

C. Growth Characteristics: Mycobacterium is obligate aerobes and derives energy from the oxidation of many simple carbon compounds and increased CO2 tension enhances growth. Biochemical activities are not characteristic, and the growth rate is much slower than that of most bacteria. The doubling time of tubercle bacilli is about 18 hours. Saprophytic forms tend to grow more rapidly well at 22-230C to produce more pigment and to be less acid- fast than pathogenic forms (Burrel, 2005). 

D. Pathogenicity of mycobacterium: There are marked differences in the ability of mycobacterium to cause lesions in various host species. Humans and guinea pigs are highly susceptible to m. tuberculosis infection, whereas fowl and cattle are more resistant. M. tuberculosis and m. bovis are equally pathogenic to humans and the route of infection determines the pattern of lesions (NTBLCP, 2005). 

E. Constituents of tubercle bacilli: The constituents found in cell walls of tubercle bacilli include lipids, proteins and polysaccharides. Mycobacterium cell walls can induce delayed hypersensitivity and some resistance to infection and can replace whole mycobacteria cells in freunds adjuvant. Mycobacterium cell wall contents only elicit delayed hypersensitivity reactions in previously sensitized animals. 

F. Pathogenesis: Mycobacterium is a droplet of 1-5µm in diameter when inhaled and reach alveoli. The disease results from establishment of virulent organisms and interactions with the host. (Bayer, 1995). Body developes certain level of cell mediated immunity that protects the individual. This immunity is destroyed by HIV/AIDS, leading to reactivation hence increase of cases. Destruction of lung parenchyma and cavitation (tubercle) is important feature of adult tuberculosis. There is also cell death, necrosis. (K. Parks, 2009. 

 G. Pathology: The production and development of lesions and their healing or progression are determined mainly by:  
(1). the number of mycobacterium in the inoculums and the subsequent multiplication.  
(2). the resistance and hypersensitivity of the host (Allewelt, 2002). 

H. Immunity and hypersensitivity: Unless a host dies during the first infection with tubercle bacilli, a certain resistance is acquired and there is an increased capacity to localize tubercle bacilli, retard their multiplication, limit their spread and reduce lymphatic dissemination. This can be attributed to the development of cellular immunity during the initial infection with evident ability of mononuclear phagocyte to limit the multiplication of ingested organism and even to destroy them. Antibodies form against a variety of the cellular constituents of the tubercle bacilli. The presence of antibodies can be determined by many different serologic tests. None of these serologic reactions bears any unequivocal relations to the immune state of the host, but high titers of IgG antibody is detectable by EIA test with polysaccharides which exists in many patients with active pulmonary tuberculosis. (Atlas, 1995). 

I. Clinical features: This includes coughing for up to  three months or more with or without sputum, weight loss, fever especially at nights, tiredness, chest pain,  blood in sputum etc.
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