Tinea capitis is most prevalent between 3 and 7 years of
age. It is slightly more common on boys than girl. Infection by Trichophyton
tonsurans may occur in adults.
Anthropophilic infections such as I. Tonsurans are more common in crowded living conditions.
The fungus can contaminate hairbrushes, clothing, towels and the backs of
seats. The spores are long lived and can infect another individual months
later.Baron et al (2003).
Zoophilic
infections are due to direct contact with an infected animal are not generally
passed from one person to another. Geophilic infections usually arise when
working in infected soil but are sometimes transferred from an infected animal.Tinea
capitis may present in several ways Anosike et al (2005).
Dry Scaling: like dandruff
but usually moth-eaten hair loss.
Black dots: The hair are
broken off at the scalp surface, which is scaly smooth areas of hair loss
kerion very inflamed mass, like an abscess.
Favus: Yellow crusts and
matted hair carrier state no symptoms and only mild scaling (T.tonsurans).
Tinea
capitis may result in swollen lymph
gland at the sides of the back of the neck. Untreated kerior and favus may result in permanent scarring
(bald areas).
In
some countries, infected children are not allowed to attained school. Elsewhere
children with Tinea capitis can attend school providing they are
receiving treatment. Carriers many have no symptoms Omar (2000).
REFERENCES
Faith, H.l, Al-
samarai, A.G.M. (2000).Prevalrnce of tinea capitis among school children
on Irag. Eastern Mediterranean Health journal. 6(1): 128 – 137.
Figueroa, J.I.
(1997). Tinea capitis in south western Ethiopia: a study of risk factors
for infection and carriage, international Journal Dermatology. 36, 661 –
666.
Fisher.F, Cook,
N.B. (1998). Fundamentals of diagnostic mycology. Phladelpslria, WB sauders
company, 156pp.
Fox, t.c (1994)
Further contribution to the
study of
the endothrix Trichophyta Flora
in London. Proc. Roy.
Med. 2,1.
Higgins, E. M,
Frller, L. C, Smith, C.H. (2000) Tinae capitis. Goudehines for the
management of Tinea capitis – British Association Dermatologist 6, 1-5.
Ive, F.A; (1966).
The carrier stage of tinea capitis in Nigeria. British Journal
Dermatology. 78 (4), 219 – 221.
Jawetz M.A Brooks
G.F., Butel J.S., Morse M.A.
(1998). medical microbiology.
(23rd ed.) McGraw –Hill
Education Press. Asia 629 - 632 pp
Kern, M.E; (1985).
Medical Mycology phladophria F. A. Dans company. 64pp.
Mercantini, R,
Marsela, R, Caprilla, F; (1978).Isolation of keratomyates from the soil of
could animal cages and endosmes in the zoo of the parco nazionaled Abruzzo,
Italy. Sabonraudia 16, 285- 259.
Mercantini, R,
Marcella, R, Caprilli, F, Idovgiallo, G. (1980) Isolation of keratinophilic
fungi from floors in Roman primary schools.mypathologia 82. 115- 120.
Ogbonna, C.I.C,
Robinson, R.O, Abubakar, J.M . (1985).The distribution of ringworm
infections among primary school children in jos, plateau state of Nigeria.
Mycopathologia 89, 101- 106.
Omar, A. A. (2000).
Ringworm of the scalp in primary school children in Alenandria: infection and
carriage. Eastern Mediteranean Health Journal. 6(5): 961- 967.
Rippon, J. W.
(1974). Medical Mycology: The pathogenic fungi and pathogenic Actinomycetes.
W.B. sunders, C.phsadephia London Toronto, 74pp.
Temple M.E, Staats
C.C Korstanje M.J. (1999).
Fungal infection in the Netherlands
prevailing fungi and pattern infection dematol. 190: 39 -42
Venugopal, P.V,
Venugopal, T.V. (1993). Tinea capitis in sandi Arania, int. journal
Dermatologist. 32: 39 – 40.
Weary,P.E. (1968)
Pityrosporum ovale. Observations on some aspects of host- parasite
interrelationship. Archs. Desm. 98, 408.