EXPLANATION OF LEUCOCYTES

The leucocytes are cells of the immune system which are involved in the defense against both infectious diseases and foreign materials (Lafleur-Brooks; 2008). They are derived from the puripotent stem cell in the bone marrow known as the hematopoietic stem cells. The neutralization, killing and poisoning of the germs is possible in the human system because of the involvement of WBC; these cells can  be induced to produce antibodies with the help of vanccine
thus preparing the body for defense.

They are also called leucocytes because they have no haemoglobin, which makes them appear colourless and are found throughout the body including the blood and the lymphatic system (Maton et al; 1000008). They are nucleated and amoeboid; the amoeboid nature of these cells helps them to squeeze through the wall of the blood vessels in order to engulf bacteria. These cells can be divided into two main types; the granulocytes (containing grnules) such as the neutrophils, eosinophils and basophils and the other type called the aggranulocytes and monocytes. 

The numbers of white cells are normally between 4-110 x 109/L white blood cells in a liter of blood making it up to 1% in a healthy adult (Albert;2005). White blood cell counts below or above 4.0 x 109/L or 11.109/L is called leucopaenia and leucocytosis respectively and the physical properties of leucocytes such as volume, conductivity and granularity may be due to activation, presence of immature cells or malignant leucocytes which leads to leukaemia.

REFERENCE 
Green W. (2008). An Introduction to Indoor Air Quality: carbon monoxide, United States Environmental protection agency. Retrieved 2008-12-16.
Gosink T. (1983). “What Do Carbon Monoxide Levels Mean?”. Alaska Science Forum. Geophysical Institute, University of Alaska Fairbanks.
Haematocrit (HCT) or packed Cell Volume (PCV). DoctorsLunge.com.Retrieved 2007-12-26.
Hardy K.R. and Thom S.R. (1994).”Pathophysiology and treatment of carbon monoxide poisoning”. Journal of Toxicology. Clinical Toxicology 32 (6): 613-629.
Hampson N.B. (1998). “Emergency department visits for CO poisoning in htre Pacific Northwest”. The Journal of emergency medicine 16: (5): 695-698.
Haynes W.M. (2010); Handbook of chemistry and physics (91 edition). Boca Raton Florida: CRC Press pp 9-39.
Henrik I. And Leif H.(2009). Relationship between cigarette smoking and high PCV and HGB levels. Scandanavian Journal of Haematology. Vol 8:241-244.
Henry J.A. (1999): Carbon monoxide, Journal of accident and emergency medicine 1999;16:91-96.
Hester V.V., Dirkje S.P., Wim Timens, Machteld NH Brigitte W.M. (2005); acute effects of cigarette smoking on inflammation in healthy intermittent smokers.
Jaffe and Badman (1995); automotive exhaust chemical: disease causing effects. Jeffery P.K. and Reid L.M. (1981); the effect of tobacco smoke with or without phenylmethyloxadiazole (PMO), on rat bronchi8al epithelium: a light and electron microscopic study. J Pathol 1991;133:341-359.
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